Anesthesia

Emergence from anesthesia

Emergence from anesthesia has cardiorespiratory, metabolic/endocrine and neurologic consequences. Awakening and extubation after anesthesia are associated with haemodynamic arousal lasting 10-25 minutes. This activation is partially mediated by elevations in catecholamine levels and partially by nociceptive stimuli. The aim of emergence after a neurosurgical procedure is to maintain intracerebral and extra-cerebral homeostasis. This implies maintenance

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Trilene anesthesia

Trilene anesthesia is a non-flammable, good analgesic-and cheap agent. Physical properties of trilene anesthesia : It has bp 80°C, VP 60 mm Hg, oil/gas coefficient 600 and blood/gas 12, metabolism 40%, unstable, preservative 1:10,000 thymol as stabilizer and coloured with waxoline blue 1 in 200,000, MAC 0.17%. Trilene anesthesia reacts with sodalime to produce phosgene

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Isoflurane respiratory effects

One of the Isoflurane respiratory effects is that, Isoflurane causes bronchodilatation by inhibiting bronchoconstriction. As with other potent vapours, isoflurane impairs both ventilatory drive and hypoxic pulmonary vasoconstriction leading to hypercapnia and V/Q mismatch. In asthmatic patients due to its lack of arrhythmogenic potential, drugs like aminophylline or agonists such as salbutamol may be less

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